A new study outlines the biological role of TREM-1 as a master upstream amplifier of innate immunity, highlighting its synergy with Toll-like receptors and its emergence as a high-value therapeutic target for stopping acute sepsis, inflammatory arthritis, and microglial-driven neurodegeneration.
A new study outlines the biological role of TREM-1 as a master upstream amplifier of innate immunity, highlighting its synergy with Toll-like receptors and its emergence as a high-value therapeutic target for stopping acute sepsis, inflammatory arthritis, and microglial-driven neurodegeneration.
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